Fuzzy linguistic prediction model for sinoatrial node field potential analysis in acute hyperglycemia environment.
Feng Y, Cao H, Wang Y, Zhang Y.
Biomed Mater Eng. 2015 Aug 17;26 Suppl 1:S881-7

Prediction model of sinoatrial node field potential using high order partial least squares.
Feng Y, Cao H, Zhang Y.
Biomed Mater Eng. 2015 Aug 17;26 Suppl 1:S1805-11.

Termination of aconitine-induced atrial fibrillation by the KACh-channel blocker tertiapin: underlying electrophysiological mechanism.
Suzuki K, Matsumoto A, Nishida H, Reien Y, Maruyama H, Nakaya H.
Pharmacol. Sci., 2014 Jul, 125, 406-14.

Heart failure enhances arrhythmogenesis in pulmonary veins.
Chang SL, Chen YC, Yeh YH, Lai YJ, Yeh HI, Lin CI, Lin YK, Lin YJ, Wu TJ, Huang YK, Chen SA, Chen YJ.
Clin Exp Pharmacol Physiol. 2011 Oct;38(10):666-74.

Time-dependent block of ultrarapid-delayed rectifier K+ currents by aconitine, a potent cardiotoxin, in heart-derived H9c2 myoblasts and in neonatal rat ventricular myocytes.
Wang YJ, Chen BS, Lin MW, Lin AA, Peng H, Sung RJ, Wu SN.
Toxicol Sci. 2008 Dec;106(2):454-63.

On the mechanisms of arrhythmias in the myocardium of mXinalpha-deficient murine left atrial-pulmonary veins.
Lai YJ, Huang EY, Yeh HI,Chen YL, Lin JJ, Lin CI.
Life Sci. 2008 Aug 15;83(7-8):272-83.

Loss of mXinalpha, an intercalated disk protein, results in cardiac hypertrophy and cardiomyopathy with conduction defects.
Gustafson-Wagner EA, Sinn HW, Chen YL, Wang DZ, Reiter RS, Lin JL, Yang B, Williamson RA, Chen J, Lin CI, Lin JJ.
Am J Physiol Heart Circ Physiol. 2007 Nov;293(5):H2680-92.